SUZUKI Atsushi

Organization

Faculty of Engineering, Division of Materials Science and Chemical Engineering

Title

Associate Professor

Research Fields, Keywords

Developmental Biology



Graduating School 【 display / non-display

  • 1998.04
    -
    2002.03

    Tokyo Institute of Technology   Faculty of Life Science and Engineering   Graduated

Graduate School 【 display / non-display

  • 2002.04
    -
    2004.03

    Tokyo Institute of Technology  Graduate School, Division of Life Science and Engineering  Master Course  Completed

  • 2004.04
    -
    2007.03

    The Graduate University for Advanced Studies  Graduate School, Division of Life Science  genetics  Doctor Course  Completed

Degree 【 display / non-display

  • Ph.D -  The Graduate University for Advanced Studies

Field of expertise (Grants-in-aid for Scientific Research classification) 【 display / non-display

  • Integrative animal science

  • Developmental biology

  • Cell biology

 

Papers 【 display / non-display

  • Essential role of mouse Dead end1 in the maintenance of spermatogonia.

    Niimi Y, Imai A, Nishimura H, Yui K, Kikuchi A, Koike H, Saga Y, Suzuki A.

    Developmental Biology   445 ( 1 )   103 - 112   2019.01  [Refereed]

    Joint Work

    Web of Science DOI PubMed

  • Requirement of the 3'-UTR-dependent suppression of DAZL in oocytes for pre-implantation mouse development.

    Fukuda K, Masuda A, Naka T, Suzuki A, Kato Y, Saga Y.

    PLoS Genetics     2018.06  [Refereed]

    Joint Work

    Web of Science DOI PubMed

  • Specific intron-dependent loading of DAZAP1 onto the cox6c transcript suppresses pre-mRNA splicing efficacy and induces cell growth retardation.

    Sasaki K, Ono M, Takabe K, Suzuki A, Kurihara Y.

    Gene.   657   1 - 8   2018.05  [Refereed]

    Joint Work

    Web of Science DOI PubMed

  • NEDD4 controls spermatogonial stem cell homeostasis and stress response by regulating messenger ribonucleoprotein complexes.

    Zhou Z, Kawabe H, Suzuki A, Shinmyozu K, Saga Y.

    Nature Communications   8 ( 15662 )   2017.04  [Refereed]

    Joint Work

    Web of Science DOI PubMed

  • Dead end1 is an essential partner of NANOS2 for selective binding of target RNAs in male germ cell development.

    Suzuki A, Niimi Y, Shinmyozu K, Zhou Z, Kiso M, Saga Y.

    EMBO reports   17 ( 1 )   37 - 46   2016.01  [Refereed]

    Joint Work

     View Summary

    NANOS2結合タンパク質としてDead end1を同定し、その結合が生殖細胞の発生に非常に重要であることを示した。

    Web of Science DOI PubMed

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Grant-in-Aid for Scientific Research 【 display / non-display

  • Grant-in-Aid for Young Scientists(A)

    Project Year: 2017.04  -  2021.03 

  • Grant-in-Aid for Scientific Research on Innovative Areas

    Project Year: 2016.04  -  2018.03 

  • Grant-in-Aid for Scientific Research(C)

    Project Year: 2014.01  -  2016.03 

  • Grant-in-Aid for Scientific Research on Innovative Areas

    Project Year: 2014.01  -  2015.03 

  • Grant-in-Aid for Scientific Research on Priority Areas

    Project Year: 2011.04  -  2012.03 

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Presentations 【 display / non-display

  • Establishment of the live imaging system of testicular teratoma in organ culture method

    2018.11  

  • Testicular teratoma in organ culture system

    Nishimura H., Imai A., Niimi Y., Suzuki A.

    (Kyusyu University, Fukuoka, Japan)  2017.07  

  • Mouse Dead end1 represses the acquisition of pluripotency in male PGCs.

    Niimi Y., Suzuki A.

    The International Research Symposium on Regulation of Germ Cell Development in vivo and in vitro  (Kyusyu University, Fukuoka, Japan)  2017.07  

  • Functional analysis of mouse Dead end1 in the maintenance of spermatogonial cells.

    Niimi Y., Kikuchi A., Tazu Y., Suzuki A.

    The 2016 meetings on GERM CELLS  (Cold Spring Harbor Laboratory, New York)  2016.10  

  • Mouse Dead end1 represses the acquisition of pluripotency in male PGCs.

    Niimi Y., Suzuki A.

    The 2016 meetings on GERM CELLS  (Cold Spring Harbor Laboratory, New York)  2016.10  

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